Psychiatric Comorbidity in Binge-Eating Disorder as a Function of Smoking History.[CME]
J Clin Psychiatry 2006;67(4):594-599
© Copyright 2015 Physicians Postgraduate Press, Inc.
Purchase This PDF for $40.00
If you are not a paid subscriber, you may purchase the PDF.
(You'll need the free Adobe Acrobat Reader.)
Receive immediate full-text access to JCP. You can subscribe to JCP online-only ($86) or print + online ($156 individual).
With your subscription, receive a free PDF collection of the NCDEU Festschrift articles. Hurry! This offer ends December 31, 2011.
If you are a paid subscriber to JCP and do not yet have a username and password, activate your subscription now.
As a paid subscriber who has activated your subscription, you have access to the HTML and PDF versions of this item.
Click here to login.
Did you forget your password?
Still can't log in? Contact the Circulation Department at 1-800-489-1001 x4 or send email
Objective: To examine the comorbidity of
psychiatric disorders in obese women with binge-eating disorder (BED) as a function of
Method: A consecutive series of 103
obese treatment-seeking women with current DSM-IV diagnoses of BED were administered
structured diagnostic interviews to assess all DSM-IV Axis
I psychiatric disorders. Participants were
classified as "never" or "daily" smokers, and lifetime rates
of comorbid psychopathology were compared across smoking groups using logistic
regression. The study was conducted from February 2003
to March 2005.
Results: Smokers were significantly more
likely to meet criteria for co-occurring diagnoses of
major depressive disorder (p = .03), panic disorder (p = .01), posttraumatic stress disorder
(p < .05), and substance abuse or dependence (p = .01).
Even after excluding participants with substance use
disorders, significant differences remained, with
lifetime smokers having significantly higher rates
of co-occurring anxiety disorders.
Conclusions: It is possible that for some
obese women with BED, binge eating and cigarette smoking share common functions, i.e., both
behaviors may serve to modulate negative affect
and/or anxiety. Although the current findings are consistent with a view of a common diathesis
for the development of impulsive eating, cigarette
or other substance use, and additional Axis I
psychopathology, prospective longitudinal studies
are needed to elucidate the nature of potential